1.
Effect of water-soluble PM10 on the production of TNF-α by human monocytes and induction of apoptosis in A549 human lung epithelial cells. Ghasemi Dehcheshmeh M, Ghadiri A, Rashno M, Assarehzadegan MA, Khodadadi A, Goudarzi G.
J Environ Health Sci Eng. 2021 Jan 7;19(1):143-150. doi: 10.1007/s40201-020-00588-4. eCollection 2021 Jun.
PMID: 34150225
2.
Air pollution and life expectancy in Europe: Does investment in renewable energy matter? Rodriguez-Alvarez A.
Sci Total Environ. 2021 Jun 16;792:148480. doi: 10.1016/j.scitotenv.2021.148480. Online ahead of print.
PMID: 34153769
3.
Exposure to ambient air pollution during trimesters of pregnancy and childhood allergic diseases in Wuhan, China. Guo M, Wei L, Yan H, Duan Z, Niu Z, Xiao C.
Int J Environ Health Res. 2021 Jun 22:1-11. doi: 10.1080/09603123.2021.1929873. Online ahead of print.
PMID: 34155935
4.
Investigating the relationship between particulate matter and inflammatory biomarkers of exhaled breath condensate and blood in healthy young adults. Seifi M, Rastkari N, Hassanvand MS, Naddafi K, Nabizadeh R, Nazmara S, Kashani H, Zare A, Pourpak Z, Hashemi SY, Yunesian M.
Sci Rep. 2021 Jun 21;11(1):12922. doi: 10.1038/s41598-021-92333-6.
PMID: 34155256
5.
When particulate matter strikes cities: Social disparities and health costs of air pollution. Giaccherini M, Kopinska J, Palma A.
J Health Econ. 2021 Jun 5;78:102478. doi: 10.1016/j.jhealeco.2021.102478. Online ahead of print.
PMID: 34161900
1.
Prenatal exposure to traffic-related air pollution and glucose homeostasis: A cross-sectional study. Heydari H, Najafi ML, Akbari A, Rezaei H, Miri M.
Environ Res. 2021 Jun 15:111504. doi: 10.1016/j.envres.2021.111504. Online ahead of print.
PMID: 34144009
2.
Association between short-term exposure to air pollution and COVID-19 hospital admission/mortality during warm seasons. Khorsandi B, Farzad K, Tahriri H, Maknoon R.
Environ Monit Assess. 2021 Jun 18;193(7):426. doi: 10.1007/s10661-021-09210-y.
PMID: 34142254
3.
Metagenomic characterization of indoor dust fungal associated with allergy and lung inflammation among school children. Isa KNM, Jalaludin J, Elias SM, Than LTL, Jabbar MA, Saudi ASM, Norbäck D, Hashim JH, Hashim Z.
Ecotoxicol Environ Saf. 2021 Jun 17;221:112430. doi: 10.1016/j.ecoenv.2021.112430. Online ahead of print.
PMID: 34147866
4.
Association between Meniere’s disease and air pollution in South Korea. Lee DH, Han J, Jang MJ, Suh MW, Lee JH, Oh SH, Park MK.
Sci Rep. 2021 Jun 23;11(1):13128. doi: 10.1038/s41598-021-92355-0.
PMID: 34162905
1.
Inflammatory and coagulatory markers and exposure to different size fractions of particle mass, number and surface area air concentrations in the Swedish hard metal industry, in particular to cobalt. Andersson L, Hedbrant A, Persson A, Bryngelsson IL, Sjögren B, Stockfelt L, Särndahl E, Westberg H.
Biomarkers. 2021 Jun 15:1-38. doi: 10.1080/1354750X.2021.1941260. Online ahead of print.
PMID: 34128444
2.
Acute cardiovascular effects of controlled exposure to dilute Petrodiesel and biodiesel exhaust in healthy volunteers: a crossover study. Unosson J, Kabéle M, Boman C, Nyström R, Sadiktsis I, Westerholm R, Mudway IS, Purdie E, Raftis J, Miller MR, Mills NL, Newby DE, Blomberg A, Sandström T, Bosson JA.
Part Fibre Toxicol. 2021 Jun 14;18(1):22. doi: 10.1186/s12989-021-00412-3.
PMID: 34127003
1.
Particulate matter pollution associated with schizophrenia hospital re-admissions: a time-series study in a coastal Chinese city. Ji Y, Liu B, Song J, Pan R, Cheng J, Su H, Wang H.
Environ Sci Pollut Res Int. 2021 Jun 11. doi: 10.1007/s11356-021-14816-3. Online ahead of print.
PMID: 34115296
2.
An impact of air pollution on moderate to severe relapses among multiple sclerosis patients. Elgabsi M, Novack L, Yarza S, Elgabsi M, Shtein A, Ifergane G.
Mult Scler Relat Disord. 2021 May 25;53:103043. doi: 10.1016/j.msard.2021.103043. Online ahead of print.
PMID: 34126372
3.
The COVID-19 incidence in Italian regions correlates with low temperature, mobility and PM10 pollution but lethality only with low temperature. Carta MG, Minerba L, Demontis R, Orrù G, Romano F, Scano A, Restivo A, Del Giacco S, Deidda S, Firnu D, Campagna M, Meloni F, Cossu G, Sancassiani F, Chessa L, Kalcev G, Littera R, Zorcolo L, Aviles-Gonzale CI, Usai P.
J Public Health Res. 2021 Jun 7. doi: 10.4081/jphr.2021.2303. Online ahead of print.
PMID: 34121380
4.
Association of Exposure to Particulate Matters and Multiple Sclerosis: A Systematic Review and Meta-Analysis. Lotfi F, Mansourian M, Mirmoayyeb O, Najdaghi S, Shaygannejad V, Esmaeil N.
Neuroimmunomodulation. 2021 Jun 16:1-7. doi: 10.1159/000516559. Online ahead of print.
PMID: 34134109
2019
Ozone Pollution: A Major Health Hazard Worldwide.
Zhang JJ, Wei Y, Fang Z.
Front Immunol. 2019 Oct 31;10:2518. doi: 10.3389/fimmu.2019.02518. eCollection 2019. Review.
PMID: 31736954 [PubMed – in process]
1.
Association between moderated level of air pollution and fetal growth: the potential role of noise exposure. Mariet AS, Bernard N, Pujol S, Sagot P, Thiriez G, Riethmuller D, Boilleaut M, Defrance J, Houot H, Parmentier AL, Benzenine E, Mauny F, Quantin C.
Sci Rep. 2021 May 27;11(1):11238. doi: 10.1038/s41598-021-90788-1.
PMID: 34045628
2.
PM2.5 Pollution Strongly Predicted COVID-19 Incidence in Four High-Polluted Urbanized Italian Cities during the Pre-Lockdown and Lockdown Periods. Kotsiou OS, Kotsios VS, Lampropoulos I, Zidros T, Zarogiannis SG, Gourgoulianis KI.
Int J Environ Res Public Health. 2021 May 11;18(10):5088. doi: 10.3390/ijerph18105088.
PMID: 34064956
3.
Environmental Pollution and Peripheral Artery Disease. Serra R, Abramo A, Ielapi N, Procopio S, Marino
Risk Manag Healthc Policy. 2021 May 27;14:2181-2190. doi: 10.2147/RMHP.S307150. eCollection 2021.
PMID: 34079405 Free PMC article. Review.
3.
Association between fine particulate matter air pollution and acute aortic dissections: A time-series study in Shanghai, China.
Chen J, Lv M, Yao W, Chen R, Lai H, Tong C, Fu W, Zhang W, Wang C.
Chemosphere. 2019 Nov 12;243:125357. doi: 10.1016/j.chemosphere.2019.125357. [Epub ahead of print]
PMID: 31760286 [PubMed – as supplied by publisher]
4.
Fine Particulate Matter Related to Multiple Sclerosis Relapse in Young Patients. Januel E, Dessimond B, Colette A, Annesi-Maesano I, Stankoff B.
Front Neurol. 2021 May 21;12:651084. doi: 10.3389/fneur.2021.651084. eCollection 2021.
PMID: 34093398 Free PMC article.
1.
Ambient air pollutants, diabetes and risk of newly diagnosed drug-resistant tuberculosis. Song WM, Liu Y, Zhang QY, Liu SQ, Xu TT, Li SJ, An QQ, Liu JY, Tao NN, Liu Y, Yu CB, Yu CX, Li YF, Li HC.
Ecotoxicol Environ Saf. 2021 May 24;219:112352. doi: 10.1016/j.ecoenv.2021.112352. Online ahead of print.
PMID: 34044311
2.
[Relationship between urban atmospheric environment and surrounding two-dimensional and three-dimensional landscape pattern in China.]. Li DK, Liu M, Li CL, Hu YM, Wang C, Liu C.
Ying Yong Sheng Tai Xue Bao. 2021 May;32(5):1593-1602. doi: 10.13287/j.1001-9332.202105.015.
PMID: 34042353 Chinese.
1.
The effects of an air quality alert program on premature mortality: A difference-in-differences evaluation in the region of Paris. Alari A, Schwarz L, Zabrocki L, Le Nir G, Chaix B, Benmarhnia T.
Environ Int. 2021 May 18;156:106583. doi: 10.1016/j.envint.2021.106583. Online ahead of print.
PMID: 34020299
2.
Time spent outdoors and risk of myocardial infarction and stroke in middle and old aged adults: results from the UK Biobank prospective cohort. Miguet M, Venetis S, Rukh G, Lind L, Schiöth HB.
Environ Res. 2021 May 18:111350. doi: 10.1016/j.envres.2021.111350. Online ahead of print.
PMID: 34019889
3.
Breast cancer risk in relation to ambient concentrations of nitrogen dioxide and particulate matter: results of a population-based case-control study corrected for potential selection bias (the CECILE study). Lemarchand C, Gabet S, Cénée S, Tvardik N, Slama R, Guénel P.
Environ Int. 2021 May 21;155:106604. doi: 10.1016/j.envint.2021.106604. Online ahead of print.
PMID: 34030067
4.
Local synergies and antagonisms between meteorological factors and air pollution: A 15-year comprehensive study in the Sydney region. Ulpiani G, Ranzi G, Santamouris M.
Sci Total Environ. 2021 May 15;788:147783. doi: 10.1016/j.scitotenv.2021.147783. Online ahead of print.
PMID: 34029820
5.
Particulate matter emissions during field application of poultry manure – The influence of moisture content and treatment. Kabelitz T, Biniasch O, Ammon C, Nübel U, Thiel N, Janke D, Swaminathan S, Funk R, Münch S, Rösler U, Siller P, Amon B, Aarnink AJA, Amon T.
Sci Total Environ. 2021 Aug 1;780:146652. doi: 10.1016/j.scitotenv.2021.146652. Epub 2021 Mar 20.
PMID: 34030313
1.
An Italian individual-level data study investigating on the association between air pollution exposure and Covid-19 severity in primary-care setting. Pegoraro V, Heiman F, Levante A, Urbinati D, Peduto I.
BMC Public Health. 2021 May 12;21(1):902. doi: 10.1186/s12889-021-10949-9.
PMID: 33980180 Free PMC article.
2.
Effects of short-term exposure to particulate matter on emergency department admission and hospitalization for asthma exacerbations in Brescia district. Pini L, Giordani J, Concoreggi C, Zanardini E, Pini A, Perger E, Bargagli E, Di Bona D, Ciarfaglia M, Tantucci C.
J Asthma. 2021 May 12:1-11. doi: 10.1080/02770903.2021.1929310. Online ahead of print.
PMID: 33980121
3.
Association of NO2 and Other Air Pollution Exposures With the Risk of Parkinson Disease. Jo S, Kim YJ, Park KW, Hwang YS, Lee SH, Kim BJ, Chung SJ.
JAMA Neurol. 2021 May 17. doi: 10.1001/jamaneurol.2021.1335. Online ahead of print.
PMID: 33999109
4.
Effects of short- and long-term exposure to atmospheric pollution on COVID-19 risk and fatality: analysis of the first epidemic wave in Northern Italy. Ho CC, Hung SC, Ho WC.
Environ Res. 2021 May 15:111293. doi: 10.1016/j.envres.2021.111293. Online ahead of print.
PMID: 34004167
1.
Association between particulate matter pollution and the incidence of mumps in 31 provinces from China. Zhang M, Zhu Y.
Environ Sci Pollut Res Int. 2021 May 12. doi: 10.1007/s11356-021-14287-6. Online ahead of print.
PMID: 33977431
2.
Association between long-term exposure to high levels of ambient air pollution and incidence of lung cancer in a population-based cohort. Yang S, Kim OJ, Shin M, Kim WJ, Kim SY.
Environ Res. 2021 May 8:111214. doi: 10.1016/j.envres.2021.111214. Online ahead of print.
PMID: 33974841
3.
Analysis of the spatial effect of outward foreign direct investment on air pollution: evidence from China. Zhou A, Li J.
Environ Sci Pollut Res Int. 2021 May 11. doi: 10.1007/s11356-021-13960-0. Online ahead of print.
PMID: 33974206
4.
Temporal and Spatial Features of the Correlation between PM<sub>2.5</sub> and O<sub>3</sub> Concentrations in China.
Abstract
In recent years, particulate matter of 2.5 µm or less (PM2.5) pollution in China has decreased but, at the same time, ozone (O3) pollution has become increasingly serious. Due to the different research areas and research periods, the existing analyses of the correlation between PM2.5 and O3 have reached different conclusions. In order to clarify the relationship between PM2.5 and O3, this study selected mainland China as the research area, based on the PM2.5 and O3 concentration data of 1458 air quality monitoring stations, and analyzed the correlation between PM2.5 and O3 for different time scales and geographic divisions. Moreover, by combining the characteristics of the pollutants, topography, and climatic features of the study area, we attempted to discuss the causes of the spatial and temporal differences of R-PO (the correlation between PM2.5 and O3). The study found that: (1) R-PO tends to show a positive correlation in summer and a negative correlation in winter, (2) the correlation coefficient of PM2.5 and O3 is lower in the morning and higher in the afternoon, and (3) R-PO also shows significant spatial differences, including north-south differences and coastland-inland differences.
1.
Correlation between air pollution and hospitalization due to myocardial infarction.
Davoodabadi Z, Soleimani A, Pourmoghaddas A, Hosseini SM, Jafari-Koshki T, Rahimi M, Shishehforoush M, Lahijanzadeh A, Sadeghian B, Moazam E, Mohebi MB, Ezatian V, Rabiei K, Sarrafzadegan N.
ARYA Atheroscler. 2019 Jul;15(4):161-167. doi: 10.22122/arya.v15i4.1834.
PMID: 31819749 [PubMed]
2.
Elderly Mortality and Exposure to Fine Particulate Matter and Ozone.
Conclusion: In our study, an increase in the number of deaths in the elderly population in accordance with the increase in the PM2.5 and ozone was found. The association found in our study could also influence socioeconomic burden. Future studies need to be performed in regards to younger population, as well as other air pollutants.
1.
Effects of air pollution on mortality of patients with chronic kidney disease: A large observational cohort study. Jung J, Park JY, Kim YC, Lee H, Kim E, Kim YS, Lee JP, Kim H.
Sci Total Environ. 2021 Apr 30;786:147471. doi: 10.1016/j.scitotenv.2021.147471. Online ahead of print.
PMID: 33971609
2.
Effect of Environmental and Socio-economic factors on the spreading of COVID-19 at 70 cities/provinces. Ahmed J, Jaman MH, Saha G, Ghosh P.
Heliyon. 2021 May 5:e06979. doi: 10.1016/j.heliyon.2021.e06979. Online ahead of print.
PMID: 33969235 Free PMC article.
3.
Ascorbic Acid-The Little-Known Antioxidant in Woody Plants.
Abstract
Reactive oxygen species (ROS) are constantly produced by metabolically active plant cells. The concentration of ROS may determine their role, e.g., they may participate in signal transduction or cause oxidative damage to various cellular components. To ensure cellular homeostasis and minimize the negative effects of excess ROS, plant cells have evolved a complex antioxidant system, which includes ascorbic acid (AsA). AsA is a multifunctional metabolite with strong reducing properties that allows the neutralization of ROS and the reduction of molecules oxidized by ROS in cooperation with glutathione in the Foyer-Halliwell-Asada cycle. Antioxidant enzymes involved in AsA oxidation and reduction switches evolved uniquely in plants. Most experiments concerning the role of AsA have been performed on herbaceous plants. In addition to extending our understanding of this role in additional taxa, fundamental knowledge of the complex life cycle stages of woody plants, including their development and response to environmental factors, will enhance their breeding and amend their protection. Thus, the role of AsA in woody plants compared to that in nonwoody plants is the focus of this paper. The role of AsA in woody plants has been studied for nearly 20 years. Studies have demonstrated that AsA is important for the growth and development of woody plants. Substantial changes in AsA levels, as well as reduction and oxidation switches, have been reported in various physiological processes and transitions described mainly in leaves, fruits, buds, and seeds. Evidently, AsA exhibits a dual role in the photoprotection of the photosynthetic apparatus in woody plants,..
4.
Mitochondrial Bioenergetics in Brain Following Ozone Exposure in Rats Maintained on Coconut, Fish and Olive Oil-Rich Diets.
Abstract
Dietary supplementation with omega-3 and omega-6 fatty acids offer cardioprotection against air pollution, but these protections have not been established in the brain. We tested whether diets rich in omega-3 or -6 fatty acids offered neuroprotective benefits, by measuring mitochondrial complex enzyme I, II and IV activities and oxidative stress measures in the frontal cortex, cerebellum, hypothalamus, and hippocampus of male rats that were fed either a normal diet, or a diet enriched with fish oil olive oil, or coconut oil followed by exposure to either filtered air or ozone (0.8 ppm) for 4 h/day for 2 days. Results show that mitochondrial complex I enzyme activity was significantly decreased in the cerebellum, hypothalamus and hippocampus by diets. Complex II enzyme activity was significantly lower in frontal cortex and cerebellum of rats maintained on all test diets. Complex IV enzyme activity was significantly lower in the frontal cortex, hypothalamus and hippocampus of animals maintained on fish oil. Ozone exposure decreased complex I and II activity in the cerebellum of rats maintained on the normal diet, an effect blocked by diet treatments. While diet and ozone have no apparent influence on endogenous reactive oxygen species production, they do affect antioxidant levels in the brain. Fish oil was the only diet that ozone exposure did not alter. Microglial morphology and GFAP immunoreactivity were assessed across diet groups; results indicated that fish oil consistently decreased reactive microglia in the hypothalamus and hippocampus. These results indicate that acute ozone exposure alters mitochondrial bioenergetics in brain and co-treatment with omega-6 and omega-3 fatty acids alleviate some adverse effects within the brain.
5.
Red Flag for Arterial Damage? Early Evidence of a Potential Connection with Ozone.
PMID: 31845826 [PubMed – in process]
6.
Association between residential greenness and metabolic syndrome in Chinese adults.
Abstract
Background: Residing in greener areas has several health benefits, but no study to date has examined the effects of greenness on metabolic syndrome (MetS). We aimed to assess associations between residential greenness and MetS prevalence in China, and to explore whether air pollution and physical activity mediated any observed associations. Conclusions: Our findings suggest a beneficial association for residential greenness and MetS in Chinese urban dwellers, especially for participants younger than 65 years old and those with higher household income. Particulate matter with an aerodynamic diameter ≤10 μm, nitrogen dioxide and ozone, but not physical activity, may only partially mediate the association.
1.
Changes in short-lived climate pollutants during the COVID-19 pandemic in Tehran, Iran. Borhani F, Shafiepour Motlagh M, Stohl A, Rashidi Y, Ehsani AH.
Environ Monit Assess. 2021 May 8;193(6):331. doi: 10.1007/s10661-021-09096-w.
PMID: 33966107
2.
Association of short-term exposure to air pollution with depression in patients with sleep-related breathing disorders. Lo K, Chiang LL, Hsu SM, Tsai CY, Wu D, Chou CJ, Chuang HC, Liu WT.
Sci Total Environ. 2021 Apr 28;786:147291. doi: 10.1016/j.scitotenv.2021.147291. Online ahead of print.
PMID: 33965829
3.
2020 Jan 8
[Driving Factors of the Significant Increase in Surface Ozone in the Beijing-Tianjin-Hebei Region, China, During 2013-2018].
PMID: 31854910 [PubMed]
Abstract
Photochemical pollution, which is believed to be influenced by emission changes and meteorological factors, is presently quite serious in the Beijing-Tianjin-Hebei (BTH) region, China. There is a need to ascertain the effectiveness of air quality management in the region based on long-term air quality trends independent from meteorological influences. We apply Kolmogorov-Zurbenko (KZ) filtering, a technique used to separate different scales of motion in a time series, to analyze the time series of the maximum daily 8-hour running average for ozone (O3-8h) from 13 cities in the BTH region during 2013-2018, and also discuss trends and driving factors. Results of the KZ filtering revealed that the short-term, seasonal, and long-term components of the O3-8h accounted for 32.7%, 63.9%, and 3.4% of the total variance, respectively. The long-term component of the BTH region was much higher than of those reported by others for Berlin, Paris, and London, and was comparable to that of Los Angeles in the early 1990s and in the 4 years previous to our study. Although we found a lower long-term component than of those reported for Shanghai and Nanjing in the Yangtze River Delta, China, the BTH region had higher rates of increase that ranged from 2.31 to 7.12 μg·(m3·a)-1[mean 4.97 μg·(m3·a)-1]. Based on the linear fitting results-that had not been verified by experiments or model simulations-the average increase rates could be mainly attributed to emission changes (90.4%), which may be distinguished into two parts, the decrease of particulate matter (PM) (27.3%) and the emission of O3 precursors (63.1%). Decreases of PM2.5in Beijing, Langfang, Tianjin, and Hengshui were considered to be responsible for the increase at the levels of 50.8%, 32.5%, 36.7%, and 48.6%, respectively. This suggests that the rapid decrease in PM2.5 could be the most important factor in the increasing trend of O3 in some cities. We conclude that further decreases in the emission of O3 precursors are required to overcome the effect of decreasing PM2.5 causing an increase in O3.
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